Herceptin Benefits May Extend Beyond Breast Cancer

March15, 2001

The basis for a breakthrough "guided missile" therapy for breast cancer, first tested in a human at the UAB Comprehensive Cancer Center, is touted in this week's New England Journal of Medicine (NJME) as having a high potential for treating other kinds of cancer.

A new UAB professor, Matthias H. Kraus, MD, was a part of the effort that originally identified the genetic target for Herceptin. In 1999 the drug became the first monoclonal antibody treatment to receive government approval for cancer treatment. Monoclonal antibodies provide a highly tailored treatment approach as opposed to broader, systemic drug treatments that generally cause more side effects. The Cancer Center has a long history of interest in monoclonal antibodies. The center participated in the original clinical trials of the drug, enrolling a Houston TX woman as the very first participant in the study.

The prestigious NEJM recently published the first study that favorably compares a combination of Herceptin and chemotherapy with chemotherapy alone in women whose tumors produce an excess of ErbB/EGFR (epidermal growth factor-receptor). Over-expression of the growth factor receptor is associated with higher aggressiveness of breast tumors.

Previous studies compared Herceptin alone against conventional therapy. Herceptin, known generically as trastuzumab, is a genetically engineered molecule that blocks the aberrant receptor activity involved in growth that is characteristic of cancer. Some 25 to 30% of breast cancer patients exhibit this genetic characteristic.

In an accompanying editorial, the NEJM said the study is "the beginning of an important new era in cancer treatment, since many more such targeted therapies are now undergoing clinical evaluation." Dr. Kraus, who recently joined the Cancer Center as an Avon Breast Cancer Scholar, was a scientist at the National Institutes of Health laboratory that identified the molecule (called ErbB2 or HER2) as one of the receptors that eventually became Herceptin's target.

Dr. Kraus said, "My research continues to focus on genetic control of transformation with particular refrence to functional aspects of signal transduction by ErbB receptor tyorsine kinases in transformation and cancer." Formerly director of the Milan Cancer Institute, he was recruited to UAB as one of a handful of new faculty made possible by a gift last year from the Avon Products Foundation. Dr. Kraus said he was attracted to UAB and the Cancer Center by their strong history in translational medicine — "bringing basic research discoveries to clinical application."

Mansoor Saleh, MD, UAB Cancer Center associate director of clinical networks, directed the center's participation in the first clinical trial of Herceptin and was an investigator in the study featured in NEJM. Also identified as an investigator in the publication today was M. Schreeder, MD, of Huntsville, who is part of the UAB cancer trials network.

UAB Health System
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